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ras gtpase inhibitor 1  (TargetMol)


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    TargetMol ras gtpase inhibitor 1
    Ras Gtpase Inhibitor 1, supplied by TargetMol, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/ras+gtpase+inhibitor+1/RAS+GTPase+inhibitor+1/pmc08631275-35-2-12
    Average 90 stars, based on 1 article reviews
    ras gtpase inhibitor 1 - by Bioz Stars, 2026-10
    90/100 stars

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    other:

    Article Title: Clonorchis sinensis Granulin Promotes Malignant Transformation of Hepatocyte Through EGFR-Mediated RAS/MAPK/ERK and PI3K/Akt Signaling Pathways
    Article Snippet: RAS inhibitor, RAS GTPase inhibitor 1 (No. # T12692), was purchased from Topscience (China).



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    miR-373 activates the PI3K/AKT–Rac1–JNK pathway of the MG-63 cell line. (A) The effect of aberrant miR-373 on the PI3K/AKT pathway of the MG-63 cell line. (B) The effect of aberrant miR-373 on Rac1 and JNK of the MG-63 cell line. (C) The effect of overexpressing miR-373 on the PI3K/AKT–Rac1–JNK pathway with the PI3K/AKT inhibitor LY294002. (D) The effect of overexpressing miR-373 on the PI3K/AKT–Rac1–JNK pathway with the Rac1 inhibitor <t>NSC23766.</t> miR, microRNA; GAPDH, glyceraldehyde-3-phosphate dehydrogenase; PI3K, phosphoinositide 3-kinase; GTP, guanosine triphosphate; Rac1, Ras-related C3 botulinum toxin substrate 1; JNK, c-Jun N-terminal kinase.
    Ras Related C3 Botulinum Toxin Substrate 1 (Rac) Guanosine Triphosphate (Gtpase) Nsc23766 Inhibitor, supplied by Millipore, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    ras gtpase inhibitor 1  (Topscience Co Ltd)
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    miR-373 activates the PI3K/AKT–Rac1–JNK pathway of the MG-63 cell line. (A) The effect of aberrant miR-373 on the PI3K/AKT pathway of the MG-63 cell line. (B) The effect of aberrant miR-373 on Rac1 and JNK of the MG-63 cell line. (C) The effect of overexpressing miR-373 on the PI3K/AKT–Rac1–JNK pathway with the PI3K/AKT inhibitor LY294002. (D) The effect of overexpressing miR-373 on the PI3K/AKT–Rac1–JNK pathway with the Rac1 inhibitor <t>NSC23766.</t> miR, microRNA; GAPDH, glyceraldehyde-3-phosphate dehydrogenase; PI3K, phosphoinositide 3-kinase; GTP, guanosine triphosphate; Rac1, Ras-related C3 botulinum toxin substrate 1; JNK, c-Jun N-terminal kinase.
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    ras gtpase inhibitor 1  (TargetMol)
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    TargetMol ras gtpase inhibitor 1
    miR-373 activates the PI3K/AKT–Rac1–JNK pathway of the MG-63 cell line. (A) The effect of aberrant miR-373 on the PI3K/AKT pathway of the MG-63 cell line. (B) The effect of aberrant miR-373 on Rac1 and JNK of the MG-63 cell line. (C) The effect of overexpressing miR-373 on the PI3K/AKT–Rac1–JNK pathway with the PI3K/AKT inhibitor LY294002. (D) The effect of overexpressing miR-373 on the PI3K/AKT–Rac1–JNK pathway with the Rac1 inhibitor <t>NSC23766.</t> miR, microRNA; GAPDH, glyceraldehyde-3-phosphate dehydrogenase; PI3K, phosphoinositide 3-kinase; GTP, guanosine triphosphate; Rac1, Ras-related C3 botulinum toxin substrate 1; JNK, c-Jun N-terminal kinase.
    Ras Gtpase Inhibitor 1, supplied by TargetMol, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/ras+gtpase+inhibitor+1/RAS+GTPase+inhibitor+1/pmc08631275-35-2-12
    Average 90 stars, based on 1 article reviews
    ras gtpase inhibitor 1 - by Bioz Stars, 2026-10
    90/100 stars
    		  Buy from Supplier

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    miR-373 activates the PI3K/AKT–Rac1–JNK pathway of the MG-63 cell line. (A) The effect of aberrant miR-373 on the PI3K/AKT pathway of the MG-63 cell line. (B) The effect of aberrant miR-373 on Rac1 and JNK of the MG-63 cell line. (C) The effect of overexpressing miR-373 on the PI3K/AKT–Rac1–JNK pathway with the PI3K/AKT inhibitor LY294002. (D) The effect of overexpressing miR-373 on the PI3K/AKT–Rac1–JNK pathway with the Rac1 inhibitor NSC23766. miR, microRNA; GAPDH, glyceraldehyde-3-phosphate dehydrogenase; PI3K, phosphoinositide 3-kinase; GTP, guanosine triphosphate; Rac1, Ras-related C3 botulinum toxin substrate 1; JNK, c-Jun N-terminal kinase.

    Journal: Oncology Research

    Article Title: MicroRNA-373 Promotes Growth and Cellular Invasion in Osteosarcoma Cells by Activation of the PI3K/AKT–Rac1–JNK Pathway: The Potential Role in Spinal Osteosarcoma

    doi: 10.3727/096504016X14813867762123

    Figure Lengend Snippet: miR-373 activates the PI3K/AKT–Rac1–JNK pathway of the MG-63 cell line. (A) The effect of aberrant miR-373 on the PI3K/AKT pathway of the MG-63 cell line. (B) The effect of aberrant miR-373 on Rac1 and JNK of the MG-63 cell line. (C) The effect of overexpressing miR-373 on the PI3K/AKT–Rac1–JNK pathway with the PI3K/AKT inhibitor LY294002. (D) The effect of overexpressing miR-373 on the PI3K/AKT–Rac1–JNK pathway with the Rac1 inhibitor NSC23766. miR, microRNA; GAPDH, glyceraldehyde-3-phosphate dehydrogenase; PI3K, phosphoinositide 3-kinase; GTP, guanosine triphosphate; Rac1, Ras-related C3 botulinum toxin substrate 1; JNK, c-Jun N-terminal kinase.

    Article Snippet: Thereafter, cells were exposed to the phosphoinositide 3-kinase (PI3K) LY294002 inhibitor (10 μM; Sigma-Aldrich, St. Louis, MO, USA) or the Ras-related C3 botulinum toxin substrate 1 (Rac) guanosine triphosphate (GTPase) NSC23766 inhibitor (50 μM; Calbiochem, San Diego, CA, USA) for 24 h.

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